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This raises the issue as to whether centrally acting leptin and MTII improve cardiac function in these hearts, and whether any effects on mitochondrial oxidative metabolism are simply due to a better functioning heart

Supporting this hypothesis, mutations that interfere with the proximal CTD-core interactions cause a robust increase in activity that is not further stimulated by G q

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Lowering the dosage or splitting it into smaller doses throughout the day may help alleviate these symptoms

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