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Zehethofer et al., 2015), the regulatory effects of FAO and CPT on COPD pathogenesis remains unclear ( CS exposure has been reported to promote FAO and mitochondrial respiration, along with an increased expression of CPT1 in the airway epithelial cells (EpiCs) ( in vitro and in vivo (Shapiro and Ingenito, 2005), which could subsequently promote FAO in EpiC (Schafer et al., 2009)

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Recent studies have reported that lipids from neighboring adipose tissues, lipoproteins, lysophospholipids, and intracellular storage fat have the potential to maintain viability and growth of cancer cells 6,7,8,9

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