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Interestingly, ABHD5 mutations causative in CDS do not co-localize to LDs and are unable to stimulate PNPLA2 lipolytic activity (30), demonstrating an important function for the interaction in the etiology of CDS and lipid storage

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Research involving BPC-157 focuses on its interactions with signaling pathways such as VEGFR2, FAK, ERK1/2, and nitric oxide-associated regulatory networks
