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First, under normal conditions, elevated HO1 expression attenuates oxidative stress to exert cytoprotective effects, and second, sustained HO1 upregulation releases ferrous iron to exacerbate oxidative stress, which leads to ROS accumulation through the Fenton reaction
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Alterations in these main receptors may lead to disorders such as hypogonadism or androgen insensitivity syndrome [59]
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