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n-3 fatty acids and cardiovascular events after myocardial infarction

To determine whether activation of Kir6.1 in cardiomyocytes was responsible for the reduction in contractile efficacy identified in Figure 9, experiments were carried out in the presence of 10 M PNU37883A to inhibit the Kir6.1 current in ventricular myocytes in the presence of 100 M TMAO

[DOI] [PMC free article] [PubMed] [Google Scholar] 75.Bonilla D.A., Kreider R.B., Stout J.R., Forero D.A., Kerksick C.M., Roberts M.D., Rawson E.S

Furthermore, Zhou et al

However, others reported that TAU could prevent atherosclerosis without significantly affecting the serum cholesterol level 14
