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doi: 10.1155/2017/4645701
A.Al-TabakhaM
Our results indicate increased DNA damage and reduced DNA repair are key targets of the dual agent treatment, and together these suggest the cancer cells are unable to repair increased DNA damage leading to higher cell death, further supporting the potential use of this dual agent as TNBC therapeutic
Available from: leistungen-tarife/hta/hta-projekte/vitaminb12tests.html Infante M, Leoni M, Caprio M, Fabbri A
APS could activate the nuclear erythroid-derived 2-related factor 2 (NRF2) pathway and its downstream protein glutamate-cysteine ligase and heme oxygenase-1 (HO-1) to anticipate anti-oxidative stress, reversing the marker enzyme of peroxidase catalase, MDA, SOD, and GSH-Px after suffering OS damage (Yan Y
