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However, the accumulated data suggest that the overactivation of this signaling system could stem from inactivation or genetic ablation of the tuberous sclerosis complex (TSC) [261, 262], the master negative regulator of mTOR

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The measurements in this study were taken from two voxels, positioned in two anatomically and functionally different parts of the ACC, its rostral portion, the pregenual ACC (pgACC) and in its dorsal portion, the dorsal ACC (dACC) [33]
