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Therefore, H 2 O 2 -induced oxidative stress recapitulates the developmental bias of GPX2 KO, and Se treatment rescues this phenotype by restoring the expression of pancreatic and liver markers in KO cells
The aspartate which is also produced is translocated out of the mitochondrion, in exchange for glutamate, through the actions of the SLC25A13 antiporter
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doi: 10.1016/j.aquaculture.2023.740327 120 HuangK.-C.LeeJ.-W.HuY.-F.BallantyneR.LiuC.-H
