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MOTS-c activates AMPK to improve metabolic function and fat oxidation without appetite suppression
Additionally, targeting cellular senescence in enteric glial cells could offer a new approach to reversing ENS dysfunction in chronic FGID patients[53]
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The move represents the most significant regulatory shift in the peptide space in years
Unappreciated biological differences between different mouse strains, such as differential sensitivity to genetic or chemically induced disease phenotypes could mislead researchers in interpreting gene or pharmacological dependent outcomes
