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Description
doi:10.1002/mgg3.1383
The ablation of Atg5 suppresses autophagy and p62 degradation, which results in the persistent activation of nuclear factor (erythroid-derived 2)-like 2 (NFE2L2) by releasing the inhibition of KEAP1 on NFE2L2
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A diarylquinoline drug active on the ATP synthase of Mycobacterium tuberculosis
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