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Description
Pain 5 , 379389 (2001)

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131 Clinically, inhibitors such as lorlatinib and larotrectinib show promise in targeting fusion-positive GBMs
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The fact that the excitatory effect of -Glu-Glu on synaptic transmission was blocked by TTX indicates that it most likely resulted from an indirect mechanism involving first the partial effect on NMDAr leading and second the endogenous synaptic release of glutamate and glycine, in turn activating both AMPAr and NMDAr ( The changes in -Glu-Glu in the extracellular medium may thus reflect the production of GSH by astroglial cells
