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Butyrate enhances erastin-induced ferroptosis of osteosarcoma cells via regulating atf3/slc7a11 pathway
doi: 10.1159/000196540
While IMCLs themselves can be benign energy stores, the specific accumulation of toxic lipid intermediates like DAGs and ceramides is central to pathology ( Concurrently, the liver avidly takes up the excess systemic FFAs, which serve as a substrate for the hepatic overproduction of triglyceride-rich VLDL ( Beyond this shared origin in lipid overflow, novel mechanisms underscore the direct crosstalk
