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By selectively disrupting key antioxidant systemssuch as inhibiting glutathione synthesis with buthionine sulfoximine (BSO), blocking thioredoxin reductase with auranofin, or directly targeting NRF2therapies can push cancer cells beyond their limited oxidative buffering capacity

129, doi:10.1039/9781847559531-00001, ISBN 978-1-84755-899-2, retrieved 2025-06-18 Journal of the American Chemical Society

It stimulates insulin release in a glucose-dependent manner, suppresses glucagon, slows gastric emptying, and signals satiety to the brain

There have been many efforts to solve this problem by use of drug carriers