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Nielsen JB, Thorolfsdottir RB, Fritsche LG, Zhou W, Skov MW, Graham SE, et al

Environmental risk factors for inflammatory bowel diseases: an umbrella review of meta-analyses

Our mechanistic understanding of how the gut microbiota transforms dietary and endogenous molecules into metabolites shared with peripheral organs and tissues in the host needs substantial expansion

Malfunctioning phagocytosis impairs the killing of bacteria that reach the lamina propria through the permeable epithelial barrier, which results in excessive Toll-like receptor stimulation, proinflammatory cytokine secretions, and activation of acquired immune responses, all of which significantly raise intestinal inflammatory reactions in genetically susceptible individuals who harbor mutations in the IBD-susceptibility gene(s) (Duboc et al., 2013)

doi: 10.1159/000448321