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Description
As previously described under the subsection of IGF-1R, the authors investigated the let-7 function in glucose metabolism using inducible let-7 transgenic mice model and found that let-7 overexpression induced glucose intolerance by targeting multiple components of the insulin-PI3K-mTOR signaling pathway including IRS-2, IGF-1R, and INSR
life 8 (3), 272277
Dement Geriatr
RhoA/Rho-kinase as a therapeutic target in asthma
Activated microglia cause superoxide-mediated release of iron from ferritin
