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Transdermal administration of Cu (II)-atsm also significantly extended survival and decreased the percentage of copper-deficient mutant Sod1 in the spinal cord (Williams et al., 2016), suggesting that the improved neurological phenotype may be related to increased Sod1 function
Sikiric P, Rucman R, Turkovic B, et al
Deoxygenation-induced and Ca(2+) dependent phosphatidylserine externalisation in red blood cells from normal individuals and sickle cell patients
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