glutathione for breast cyst Targeting Metabolism: Partner in Crime in Anticancer Therapy The Rockefeller University » Mitochondrial

Molecular Metabolism, 46, 101102

GBM, glioblastoma multiforme

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Toxicant channeling A hypothesis in which it is suggested that reactive sulfur-containing species generated from toxic cysteine S -conjugates are channeled from the active site of mitochondrial aspartate aminotransferase to -ketoglutarate dehydrogenase complex, thereby accounting for the remarkable sensitivity of this enzyme complex to inactivation in tissues/cells exposed to certain toxic cysteine S -conjugates