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Description
PPAR agonists also downregulate glutathione (GSH) levels, increasing reactive oxygen species (ROS) levels, and downregulating retinoic acid receptor-related orphan receptor t (RORt) expression, which facilitates amelioration of Th17-cell-associated immune dysregulation ( Figure 2 ) (52)
Conversely, deletion of ttll-11 nearly abolishes glutamylation in C
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BBR treatment can reduce ROS levels in endothelial cells after acute exposure to H 2 O 2 in vitro
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