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Samples were homogenized with an Ultraturrax tissue homogenizer (IKA) and stored at 80 C until further processing

One in four contained an active drug not mentioned at all

In accordance with suggested metabolic dysfunction, hypermethylation of a CpG site within DGKD, TRPC3, and two CpG sites within KCNB1 may contribute to abnormal insulin secretion

Major drawbacks of this approach include: 1) false positive signal from incomplete thiol blocking, and 2) lengthy chemical manipulations in cell lysate

In Figure 1, the theoretical calculation of the impact of GSHs artificial oxidation on GSSG rise is shown, considered their actual ratio in blood (e.g., 500:1)