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found that HAC MSCs could inhibit the activation of inositol requiring enzyme 1 by reducing the expression of glucose regulated protein 78, effectively reversing endoplasmic reticulum stress (ER) in testicular tissue, reducing the production of apoptosis factors (e.g., Capase12 and Capase3) in rat seminiferous tubules caused by ER stress, eventually exerting an effect on mitigating radiation-induced testicular tissue damage in rat

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