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The dependency on GPX4 the central lipid hydroperoxide detoxifying enzyme is not uniformly distributed across cancers, but is markedly amplified in tumors bearing specific oncogenic alterations that destabilize redox homeostasis

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Within preclinical investigative frameworks, laboratory studies may examine peptidemetal binding interactions, structural stability, solution behavior following reconstitution, and intracellular signaling responses associated with copper-dependent biochemical pathways