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TB-500 has been particularly extensively studied in cardiac repair models with foundational research demonstrating that thymosin beta-4 and TB-500 promote cardiomyocyte survival following ischaemia-reperfusion injury, stimulate cardiac progenitor cell migration into infarcted tissue, reduce infarct size, and improve cardiac function in myocardial infarction models

Studies suggest that it: Promotes fat breakdown by activating beta-3 adrenergic receptors, leading to the release of fatty acids

This might make the treatment less effective and increase the risk of cancer coming back

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Across this body of research, no significant toxicity has been reported at doses ranging from 1 g/kg to 10 mg/kg in rodent models