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High-dose Vitamin C IV therapy supports the body at a cellular level by increasing antioxidant capacity

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Ab68 may exert its anti-inflammatory effects by preventing CD38 from binding to its cognate receptor CD31, thereby reducing CD38 + immune cell adhesion to endothelium and CD31-mediated transmigration to sites of inflammation 52,56,57

As the oxidative burden on the mitochondria is lifted, these organelles can begin to shift away from inefficient anaerobic glycolysis (which produces the brain-fog-inducing lactate) and back toward efficient aerobic ATP production

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