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In healthy tissue, the peptide maintains NO homeostasis by modestly increasing endothelial NOS (eNOS) for beneficial vasodilation while suppressing inducible NOS (iNOS) that drives pathological inflammation

In a study on GHD children, both ApoA1 and HDL-cholesterol levels were found to be similar to healthy controls independently of GH replacement therapy [18, 19]

(50) reported that sepiapterin, a tetrahydrobiopterin precursor, recoupled NOS in CRC cell lines (HCT116 and HT29), restoring the tetrahydrobiopterin: dihydrobiopterin ratio, which is significantly lower in tumors than in normal tissues

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