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Description
Background: GNAO1 variants have been associated with a phenotypes of severe early infantile onset epileptic and intellectual disability (ID), with the involuntary movements, so far reported with choreoathetosis and status dystonicus (SD)
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DOI: 10.1186/1476-511X-13-121
The proposed pathophysiology involves accelerated phagocytosis during rapid bacterial death, which induces cytokine release by monocytes, producing a systemic inflammatory response
Sizing the ends: normal length of human telomeres
