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Nonetheless, in the context of brain ischemic injury, these cytokines are rapidly and extensively upregulated [19], playing roles in both pro-inflammatory and anti-inflammatory mechanisms

*The tendons become disorganised, hypervascular, and degenerative

Without adequate circulation, the injured tissue cannot receive the oxygen and nutrients necessary for repair, nor can it efficiently remove metabolic waste products

They typically have short half-lives as proteases rapidly degrade them

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