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Measurement of total antioxidant response in colorectal cancer using a novel automated method

The binding of NAPQI to mitochondrial proteins contributes to mitochondrial dysfunction (Lancaster et al., 2015) following mitochondrial GSH (mGSH) depletion, causing the generation of reactive oxygen species (ROS), oxidative stress and mitochondrial DNA damage

Conversely, the deacetylation of histones by histone deacetylases (HDAC) restricts access of transcription factors to the DNA and suppresses transcription

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S., et al., Lysyl Oxidase Induces Vascular Oxidative Stress and Contributes to Arterial Stiffness and Abnormal Elastin Structure in Hypertension: Role of p38MAPK, Antioxidants & Redox Signaling 27, no