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Description
From a clinical perspective, reducing ZnT8 transport activity or down-regulating its cellular expression is proposed as an anti-diabetogenic strategy, mimicking the protective effect of SLC30A8 haploinsufficiency in humans
doi: 10.1016/s0079-6603(02)71040-4
Protein science : a publication of the Protein Society , 6 (10), 21802187
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