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In vitro angiogenesis assays demonstrate that GHK-Cu (0.1-1 M) increases endothelial cell tubule formation by 60-90% compared to untreated controls, with effects mediated through VEGF-independent pathways involving integrin receptor activation and focal adhesion kinase signaling

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The numbers that circulate come from compounding-clinic practice and preclinical work, and theyre general protocol guidance rather than a recommendation

Without exogenous glutamine residues provided by the peptides, repair of the epidermis did not occur within the 2-week period of treatment
