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ERR agonism is mechanistically distinct in that it amplifies the transcriptional machinery for oxidative metabolism rather than directly uncoupling mitochondria or stimulating adrenergic pathways

HA@RFM@GP@SIN NPs promoted the transition from M1 to M2 macrophages and inhibited the abnormal proliferation of FLS in vitro

Epolactaene binds human Hsp60 Cys442 resulting in the inhibition of chaperone activity

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Oxidative stress in autism spectrum disorder-current progress of mechanisms and biomarkers