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Genetic variations in the activity of glutathione-S-transferase-4, a GSH-dependent enzyme that reduces 4hydroxynonenal, is a determining factor in the extent of neurodegeneration after TBI (Al Nimer et al., 2013)

The resulting damage intensifies cellular dysfunction and suppresses mitochondrial respiration, thereby aggravating the bioenergetic failure that underlies severe sepsis (Vctor et al., 2009

doi: 10.1021/tx970136j

Schmucker S, Martelli A, Colin F et al (2011) Mammalian frataxin: an essential function for cellular viability through an interaction with a preformed ISCU/NFS1/ISD11 ironsulfur assembly complex