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The pathological role of amylin in diabetes development has been extensively studied, with elevated amylin levels leading to fibril formation that may contribute to beta cell toxicity and insulin resistance

We next determined that the memory facilitating effects of intra cerebroventricularly delivered Nle-AngIV derived from its N-terminal region given that N-directed fragments as small as tetra- and tripeptides retained the ability to overcome scopolamine-induced amnesia [2,28]