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In addition, higher levels of ROS have been shown to activate death receptors via downregulation of the FLIPa protein that regulates apoptosis and functions as a key link between cell death and survival pathways (Wang et al

Consistent with the ATF4-mediated transcriptional control for GSH biosynthesis, these cells all showed higher levels of GSH and the lack of replication stress

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In this study, we focused on osteocytes, which comprise more than 95% of bone cells [18] and were recently identified as a source of LCN2 [19]

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