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Description
It supports mitochondrial and antioxidant systems, leading to better ATP production, reduced oxidative stress, and more efficient cellular repair
Adaptation to acetaminophen exposure elicits major changes in expression and distribution of the hepatic proteome
When the synthesis of GSH synthesis is blocked, the activity of GPX4 is decreased, as is the cellular antioxidant capacity, which leads to the accumulation of ROS and promotes ferroptosis (39)
cellular NADPH level is proposed as a predictor for ferroptosis susceptibility (Shimada et al., 2016)
2011;300:E76170
