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31, S213S220

& Burgess, J

However, when both parents carry defective genes, the risk of developing moderate to severe thalassemia increases significantly

Damaged melanocytes release danger-associated molecular patterns (DAMPs), including heat shock proteins (HSPs) and oxidized lipids, which activate innate immune responses and promote the recruitment of autoreactive T cells, mediated by the release of pro-inflammatory cytokines, including IL-15, IL-17, and IL-1beta [80,81]