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This research was funded in part by a National Institutes of Health General Medicine Grant (GM85024 to E.H.), a Forschungsstipendium of the Max Planck Society (to E.H.), a BBSRC CBMNet Proof-of-Concept Award (to E.H.), a National Institutes of Health Infectious Disease and Immunology Training Grant (to D.M.Z.), and a National Science Foundation Grant (1949762 to D.M.Z.)

Impact of glutathione on acute ischemic stroke severity and outcome: possible role of aminothiols redox status

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Conversely, at high concentrations, H 2 O 2 triggers cell death, primarily through cell cycle arrest and apoptosis, by overwhelming the protective buffering capacity of redox relays (Fig

Sometimes the issue is that the real driver is not being treated