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Thus, luteolins mechanisms of action on fibroblasts include inhibition of NF-B activation, which is crucial for inflammatory responses [244], and modulating MAPK and PI3K/Akt pathways to regulate cell adhesion and inflammation [245] and suppressing TGF-1 signaling, which is central to fibroblast-to-myofibroblast differentiation and the progression of fibrosis [19]

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doi: 10.7326/0003-4819-152-2-201001190-00012