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Lancet, 2012

Bacteriostatic water is the correct choice for safety and stability

doi: 10.1172/jci.insight.153079

Other mechanisms for EtOH-induced hepatic GSH depletion may involve increased efflux of GSH from the liver as observed previously [52, 53] or enhanced utilization of GSH for the detoxification of free radicals and oxidants produced as a result of EtOH exposure [17, 54-56]

In gastrectomized animals the application of the investigated agents (i.e., NaBPC157 or the reference agents before cysteamine) significantly prevented the otherwise severe duodenal lesion development noted in the control gastrectomized cysteamine rats