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knocking down ELAVL1 alone noticeably decreased proliferation, migration, and invasion, but combining ELAVL1 and GAC knockdown (which depleted both glutaminase isoforms) did provide a small further decrease in cell growth, migration and invasion compared to that with ELAVL1 knockdown alone

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Acknowledgements This study was partially supported by (1) Grant-in-Aid for Scientific Research (C) of the Ministry of Culture and Science of Japan (20590810, 23591017, 24591020, 12008507, 17877850, 17K09468, 15K09052, 24591020, 15K09052, 18K0792, 121K07943), (2) the Research Program on Intractable Diseases, from the Ministry of Labor and Welfare of Japan, and (3) grants-in-aid from the Ministry of Education, Culture, Sports, Science and Technology of Japan, (4) the Research Program from the Japan Medical Research and Development (AMED) (17824893), and (5) The branding program as a world-leading research university on intractable immune and allergic diseases supported by the Ministry of Education, Culture, Sports, Science and Technology of Japan

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Researchers observed that treatment was associated with reduced p16-positive cell populations and changes in physical function parameters in aged animals [1]